10 Foods That Offer Genuine Hope

At Hallelujah Diet we often say that self-healing is built into your body. And when we talk about your liver, it is exhibit number one. If only a third of your liver is left, it can completely regenerate itself. Even though we have caused abused our livers with our modern processed food diet, the truth is that we can heal them with a whole foods plant-based diet. Our foods indeed are medicine for our livers.

Fatty liver disease, now officially called metabolic dysfunction-associated steatotic liver disease (MASLD), has become the most common chronic liver disease on the planet. It affects roughly one in three adults worldwide. There is no pharmaceutical treatment for MASLD, and probably never will be. The medical guidelines in Europe and America all agree that lifestyle modification is the first line of treatment. Nothing else even comes close.

Basically, here’s what you need to do for fatty liver disease: Eat real foods. 

Ultra-processed foods, sugar-sweetened beverages, and white flour are the worst offenders. When we fill up on empty calories and junk food and don’t eat wholesome vegetables, healthy carbs, and plant-based protein, we damage our livers.

We see this all the time in people who come to us. People with obesity, type 2 diabetes, insulin resistance, and elevated triglycerides often have fatty liver disease as well. These are exactly the conditions that a well-designed, whole-food, plant-based diet addresses most powerfully and quickly. Doesn’t take long.

Here is what the research actually shows about the best foods for a fatty liver, and equally important, what you need to stop eating.

What Is Fatty Liver Disease, and What Causes It?

The curious thing is that fat doesn’t necessarily accumulate in your liver from fat in your diet. Fat builds up in your liver when it’s overwhelmed. It’s excess calories, especially from refined carbohydrates, added sugars, and high fructose corn syrup sweetened beverages that really does your liver in. Your liver can turn these carbohydrates into fat because they’re more compact and easier to store. 

When your liver can’t get rid of fat fast enough (or the rest of the body doesn’t really want it, which is called insulin resistance), it ends up storing it. When that fat accumulation is 5% or more of your liver weight, you get a diagnosis of fatty liver disease. 

In the early stages, fatty liver doesn’t cause any symptoms, but left untreated, it progresses to more serious inflammatory stages. The first one is called MASH (metabolic dysfunction-associated steatohepatitis), and then fibrosis, cirrhosis, and even liver cancer. If you catch it early, it’s easy to reverse the damage. At the end, not so much. 

The root cause is excess calories, which lead to insulin resistance, increased visceral fat around your organs, a disrupted gut microbiome, and diet-related inflammation. Your liver has been assaulted by your pattern of eating, so changing a single food or adding one supplement will not change the whole war against your liver. 

The Most Important Thing: The Overall Diet Pattern

Before listing individual foods, the most important thing to understand is that no single food reverses fatty liver disease. What works is a consistent dietary pattern.

Dr. M.C. Ryan and colleagues at the University of Melbourne and Sir Charles Gairdner Hospital in Australia published a small but rigorous randomized, crossover trial in the Journal of Hepatology in 2013. Twelve non-diabetic adults with biopsy-proven NAFLD were put on either a Mediterranean diet or a low-fat, high-carbohydrate control diet for six weeks, then switched. The results were striking. Even without weight loss, after six weeks on the Mediterranean diet, liver steatosis was reduced by 38% as measured by magnetic resonance spectroscopy, a gold-standard imaging technique, compared to only 7% loss on the control diet. 

What makes the Mediterranean diet so effective for the liver is what it has in common with a whole-food, plant-based diet: abundant vegetables, legumes, whole grains, nuts, seeds, and olive oil, with very little refined carbohydrate, processed food, or added sugar. The Hallelujah Diet delivers all of these elements and then some.

The biggest impact will be gained by the biggest change. But don’t discount the first little steps. They can lead to bigger changes. Just get some momentum and keep going. 

Now on to the list of foods. By emphasizing these foods, you will gain ground quickly in recovering liver health. 

10 Best Foods for Fatty Liver Disease

1. Leafy Greens (Spinach, Arugula, Spring Mix, Kale, Collards)

Leafy greens are possibly the most liver-protective food group you can eat. Leafy greens are exceptionally rich in nitrates and polyphenols, which help reduce hepatic fat accumulation and oxidative stress in the liver. 

A 2025 analysis of the NHANES survey group in 2017-2018 by Liu and coworkers was published in BMC Public Health. In this analysis, 3,162 participants were examined to see if vegetables conferred benefits for MAFLD (metabolic dysfunction-associated fatty liver disease). Only green leafy vegetables, but no other vegetables, had a significant association, reducing the odds of MAFLD by about 46%. 

Similar results were seen in a 2021 study by Li and coworkers, who studied a large Chinese adult cohort of 26,891 adults. They found a significant decrease (~28%) in risk of fatty liver disease with the highest intake of green leafy vegetables, especially among normal or overweight, but not obese, individuals. 

How to use it: Make a large salad every day using arugula, spinach, spring mix, or kale as the base. Blend dark greens into smoothies. Use collards as wraps instead of tortillas. Aim for 3 cups of raw leafy greens daily.

2. Extra Virgin Olive Oil

Most people think of fatty liver as a fat problem that requires eating less fat. The research tells a different story. The type of fat matters far more than the amount, and extra virgin olive oil is one of the most liver-protective fats available.

The key compounds are oleic acid, a monounsaturated fat that reduces hepatic lipogenesis, and a family of polyphenols, particularly oleocanthal and oleuropein, that suppress NF-kB, the same inflammatory transcription factor implicated in MASH progression. These polyphenols are what distinguish extra virgin olive oil from refined olive oil and make the quality of the oil matter significantly.

The human trial evidence is solid in olive oil’s favor. Dr. Praveen Nigam and colleagues at G.R. Medical College in Gwalior, India, published a 6-month randomized controlled trial in the American Journal of Clinical Nutrition in 2014 enrolling 93 men with ultrasound-confirmed NAFLD. Participants were randomized to use either olive oil, canola oil, or commonly used refined soybean and safflower oil as their daily cooking medium, capped at 20 grams per day. The olive oil group (and canola oil, but not control group) showed significant reductions in fatty liver grade on ultrasound: grade I prevalence dropped from 73% to 23%, grade II from 20% to 10%, and grade III from 7% to zero. Insulin resistance and triglycerides also improved significantly.

A second randomized, double-blind clinical trial by Dr. Maryam Kavyani and colleagues in Iran, published in Nutrition in 2018, enrolled 66 NAFLD patients and assigned them to either 20 grams of olive oil or sunflower oil daily for 12 weeks alongside a calorie-reduced diet. Fatty liver grade improved significantly more in the olive oil group than in the sunflower oil group, and body fat percentage declined by 3.4% in the olive oil group versus an increase of 0.38% in the sunflower oil group. The researchers concluded that olive oil reduced fatty liver severity independently of its effects on other cardiometabolic markers.

The PREDIMED trial, one of the largest dietary intervention trials ever conducted, further supports this. Among participants supplementing a Mediterranean diet with at least 4 tablespoons of extra-virgin olive oil per day, the prevalence of hepatic steatosis dropped from 33% to 8.8% over 3 years, compared with a control group advised to reduce all dietary fat.

How to use it: Use extra virgin olive oil as your primary cooking fat and salad dressing base. The range supported by the research is 2 to 4 tablespoons per day. Look for oils labeled “extra virgin” and “cold-pressed” with a harvest date on the bottle, since polyphenol content degrades significantly with age and heat processing. Refined olive oil and light olive oil do not deliver the same benefit.

3. Legumes (Lentils, Black Beans, Chickpeas, Soybeans)

Legumes work on fatty liver disease through multiple mechanisms at once. Their soluble fiber feeds gut bacteria that produce butyrate, which reduces liver inflammation. Their low glycemic index prevents the post-meal blood sugar spikes that drive insulin resistance, a core driver of fatty liver. Their plant protein replaces animal protein at meals, removing a major source of saturated fat.

A 2025 randomized controlled trial by Latypov and colleagues, published in Nature Communications, enrolled 120 adults with prediabetes and assigned them to either a legume-enriched diet or a calorie-restricted control diet. The legume group had significantly greater reductions in liver fat, LDL cholesterol, and markers of insulin resistance. The mechanism, confirmed by microbiome analysis, was a shift toward butyrate-producing gut bacteria.

How to use it: Aim for half to one cup of cooked legumes daily. Add lentils to soups, chickpeas to salads, and black beans to grain bowls. As I’ve written about in the beans and metabolic health article, even one cup per day consistently moves biomarkers in the right direction within 12 weeks.

4. Oats and Barley (Beta-Glucan Sources)

Oats and barley are the richest grain sources of beta-glucan, a soluble fiber that forms a viscous gel in the digestive tract, binding bile acids and cholesterol before they can be absorbed. This reduces the cholesterol load delivered to the liver for processing, which matters significantly in fatty liver disease because the liver is already working overtime on fat metabolism.

The human trial evidence is clear on cholesterol. Dr. Ho and colleagues published a systematic review and meta-analysis in the European Journal of Clinical Nutrition in 2016, analyzing 14 randomized controlled trials involving 615 participants. Oat beta-glucan supplementation significantly reduced LDL cholesterol and non-HDL cholesterol across the included trials. The benefit was consistent across populations and dose ranges, with a median intervention duration of four weeks, meaning the effect shows up relatively quickly.

For fatty liver specifically, the connection runs through the gut microbiome. Beta-glucan is one of the most effective prebiotic fibers available, feeding the butyrate-producing bacteria associated with reduced hepatic inflammation. This is the same butyrate pathway I described in the legumes section, and beta-glucan from oats and barley activates it just as powerfully.

How to use it: Steel-cut oats or thick traditional rolled oats make a great base for a daily breakfast. Barley works well in soups and grain bowls.

5. Flaxseeds (and Chia Seeds)

Many people claim that nuts, especially walnuts with the omega-3 fat ALA, are very beneficial for fatty liver disease, but flaxseeds outshine them altogether and deserve a place on this list. Flaxseeds contain roughly 6,400 mg of ALA per tablespoon, compared to about 2,500 mg per ounce of walnuts. Chia seeds are similarly ALA-dense. Both also deliver soluble fiber and lignans, giving them multiple mechanisms for liver benefit simultaneously.

The human trial evidence for whole flaxseed in NAFLD is genuinely solid. Dr. Zahra Yari and colleagues at Shahid Beheshti University of Medical Sciences in Tehran conducted a randomized controlled trial on 50 patients with NAFLD, assigning them to lifestyle modification alone or lifestyle modification plus 30 grams of brown milled flaxseed daily for 12 weeks. Liver enzymes, insulin resistance, hepatic fibrosis, and steatosis all decreased significantly in both groups, but the reductions were significantly greater in the flaxseed group.

A more recent 2025 RCT by Wu and colleagues, published in Food and Function, enrolled 50 NAFLD patients and measured liver fat by MRI-PDFF, the most precise non-invasive imaging method available. Patients received 30 grams of flaxseed powder daily before lunch or dinner for 12 weeks. The flaxseed group showed significantly lower liver fat content, body fat percentage, visceral fat area, AST, total cholesterol, and triglycerides after 12 weeks, along with a significant increase in HDL cholesterol. No such changes were observed in the control group.

A third randomized, double-blind controlled trial, published in the British Journal of Nutrition in 2020, tested flaxseed oil against sunflower oil in NAFLD patients following a calorie-reduced diet. Flaxseed oil did show a modest benefit on fatty liver grade and reduced IL-6 compared to sunflower oil. However, it did not replicate the more powerful improvements in liver enzymes and steatosis seen with milled flaxseed in the trials above. This is consistent with what nutritional science generally finds: the whole food, with its intact fiber, lignans, and full complement of phytochemicals, outperforms the extracted oil. If you are going to choose one form, milled flaxseed is the better option.

How to use it: One to two tablespoons of ground flaxseed daily, added to oatmeal, smoothies, or salads, delivers the dose used in the clinical trials. Strive for 30 g per day for the full benefit. Flaxseed must be ground rather than whole for ALA and lignans to be fully absorbed. Hallelujah Diet’s B-Flax-D provides stabilized ground flaxseed together with vitamins and minerals, making it a practical daily option. Chia seeds are a good second choice with a similar ALA and fiber profile. They can be added whole to smoothies, overnight oats, or salad dressings without grinding.

6. Fatty Fish and Fish Oil (Omega-3 Fats)

Most people following a plant-based diet don’t eat fatty fish regularly, and there are good reasons for that, including concerns about heavy metals, PCBs, and other persistent pollutants found in many fish. Nevertheless, the case for EPA and DHA in fatty liver disease is strong enough to warrant a direct mention, and purified fish oil is the safest way to get these long-chain omega-3 fats without the contamination risk.

EPA and DHA work in the liver through mechanisms that plant-based ALA from flaxseed cannot fully replicate. They directly suppress de novo lipogenesis, the process by which the liver converts excess carbohydrates into fat. They reduce hepatic triglyceride synthesis and improve insulin signaling in liver tissue. And they shift the fatty acid composition of the liver away from the saturated and monounsaturated fats that accumulate in NAFLD, toward more anti-inflammatory polyunsaturated forms.

The most rigorous clinical trial is the WELCOME study, conducted by Dr. Eleonora Scorletti, Dr. Philip Calder, and Dr. Christopher Byrne and colleagues at the University of Southampton, published in Hepatology in 2014. One hundred three patients with NAFLD were randomized to 4 grams per day of purified DHA plus EPA or placebo for 15 to 18 months in a double-blind, placebo-controlled design. The overall intention-to-treat analysis showed a trend toward liver fat reduction that did not reach statistical significance, partly because of variable adherence in the treatment group and some contamination in the placebo group. However, in the secondary analysis, erythrocyte DHA enrichment was independently and significantly associated with reduced liver fat, with each 1% increase in DHA tissue enrichment associated with a 1.70% decrease in liver fat percentage. The researchers concluded that substantial liver fat reductions are achievable when DHA tissue enrichment is high.

A second randomized trial by Song, Zhao, and colleagues, published in the British Journal of Nutrition in 2020, enrolled 96 NAFLD patients and tested fish oil alone (450 mg EPA plus 1,500 mg DHA daily) against placebo and phytosterol supplementation. The liver fat improvement in the fish oil group trended in the right direction but did not reach statistical significance. What did reach significance was the reduction in TGF-beta, a key driver of liver inflammation and fibrosis (P = 0.002), and TNF-alpha, a major inflammatory cytokine (P = 0.036). This matters because fibrosis is what actually kills liver patients long-term, not fat accumulation alone.

The honest summary is this: fish oil reliably lowers triglycerides and reduces liver inflammation, and shows consistent signals toward liver fat reduction. Results across RCTs are variable depending on dose, duration, and how well patients actually enrich their tissues with DHA. Getting your Omega-3 Index above 8% appears to be what separates those who benefit from those who don’t.

How to use it: Take enough purified fish oil to provide 2 to 3 grams of combined EPA and DHA daily. Testing your Omega-3 Index is the most reliable way to know whether your current dose is achieving meaningful tissue enrichment. Hallelujah Diet’s Omega-3 fish oil is purified to remove heavy metals and provides a concentrated EPA and DHA dose. For strict vegans, algae-based DHA is an alternative, though most algae oils are lower in EPA.

7. Coffee

Long before modern hepatology took an interest, alternative medicine practitioners recognized coffee as a liver remedy. Max Gerson, whose cancer therapy in the mid-twentieth century used coffee enemas as a central liver detoxification tool, wasn’t the only one. Coffee’s connection to liver health has deep roots in natural medicine traditions, and modern population science has now caught up with something that observational healers noticed decades ago.

I want to be clear that there are actually no randomized controlled trials testing whether coffee reduces liver fat in NAFLD patients. What we have is observational evidence. It’s consistent across large-scale populations and is the kind of evidence abundant in nutritional science research. It doesn’t prove causation, but it makes a strong case nevertheless. 

The best population data comes from two directions. First, the landmark analysis by Dr. Constance Ruhl and Dr. James Everhart using the Third National Health and Nutrition Examination Survey, published in Gastroenterology in 2005. Among 5,944 adults at high risk for liver injury including those with overweight, impaired glucose metabolism, and viral hepatitis, people who drank more than 2 cups of coffee per day had 44% lower odds of having elevated ALT, the primary blood marker of liver cell stress and damage. The association held consistently across every high-risk subgroup. ALT is the same marker that NAFLD patients and their doctors watch closely.

Second, for people who already have fatty liver disease, coffee appears to specifically protect against fibrosis, the scarring process that turns fatty liver into cirrhosis. A 2021 systematic review and meta-analysis by Grazioli and colleagues, published in Nutrients, pooled results from 11 studies and found that coffee drinkers with NAFLD had 35% lower odds of significant liver fibrosis compared to non-drinkers (RR 0.65, 95% CI 0.54 to 0.78), with very low heterogeneity across studies.

The active compounds responsible appear to be chlorogenic acids and diterpenes rather than caffeine itself, since both caffeinated and decaffeinated coffee show benefit in observational studies. These compounds have demonstrated anti-inflammatory and antioxidant properties that are plausible mechanisms for protecting liver tissue over time.

How to use it: Two to three cups of filtered coffee daily appears to be the range associated with liver benefit in population studies. Filtered coffee is preferable to unfiltered preparations like French press, which raise LDL cholesterol through diterpenes. Decaffeinated coffee appears to preserve the hepatoprotective benefit for those sensitive to caffeine.

8. Garlic

Garlic’s liver benefits come from its sulfur compounds, particularly allicin and S-allyl cysteine, which activate hepatic antioxidant pathways and suppress the inflammatory signals that drive MASH progression.

The clinical evidence is solid. Dr. Abbas Ali Sangouni and colleagues at Urmia University of Medical Sciences in Iran published a double-blind, randomized, placebo-controlled trial in the British Journal of Nutrition in 2020. Ninety NAFLD patients were randomized to receive either 1,600 mg of garlic powder daily (four 400 mg tablets) or placebo for 12 weeks. At the end of the trial, hepatic steatosis on ultrasound was significantly reduced in the garlic group compared to placebo. ALT dropped significantly (p < 0.001), AST dropped significantly (p = 0.002), and gamma-glutamyltransferase dropped significantly (p = 0.003). Total cholesterol, triglycerides, and LDL all improved as well.

How to use it: Fresh garlic is the most potent whole-food source. Two to four cloves per day, minced raw into dressings or added near the end of cooking to preserve allicin content, is a practical daily target. The supplement dose used in the Sangouni trial, 1,600 mg of garlic powder daily, is achievable through capsules if consistent daily cooking with garlic isn’t realistic.

9. Turmeric and Curcumin

Turmeric’s active compound curcumin is one of the most studied natural anti-inflammatory agents in liver disease, and the clinical evidence here is stronger than for most foods on this list. Multiple randomized controlled trials have now tested curcumin directly in NAFLD patients, and the results are consistently positive.

The benchmark trial is by Dr. Yunes Panahi and colleagues at Baqiyatallah University of Medical Sciences in Tehran, published in Drug Research in 2017. Eighty-seven NAFLD patients were randomized to 1,000 mg per day of phytosomal curcumin or placebo for 8 weeks. Ultrasound improvement occurred in 75% of the curcumin group versus 4.7% of the placebo group. Both AST and ALT fell significantly in the curcumin group and rose in the placebo group. BMI and waist circumference also improved.

A more recent and methodologically rigorous trial by He, Chen, and colleagues at Sun Yat-sen University, published in the American Journal of Clinical Nutrition in 2024, enrolled 80 patients with fatty liver and tested 500 mg curcumin daily for 24 weeks, the longest duration of any curcumin NAFLD trial to date. Liver fat was measured using controlled attenuation parameter (CAP) imaging, which is more precise than standard ultrasound. Curcumin significantly reduced liver fat compared to placebo (17.5 dB/m reduction, P < 0.001), along with significant reductions in triglycerides, fasting glucose, HbA1c, and insulin. The researchers also found favorable shifts in gut microbiota composition, suggesting curcumin works partly through improving bile acid metabolism in the gut.

The challenge with curcumin is absorption. Standard curcumin powder has very poor bioavailability on its own. The phytosomal and amorphous dispersion forms used in these trials are specifically engineered to improve absorption, and they are what the research supports. Hallelujah Diet’s Professional Strength Curcumin uses a CurQfen formulation with fenugreek fiber, which is one of the better-validated bioavailability-enhanced delivery systems available.

How to use it: Add turmeric generously to curries, soups, and roasted vegetables, and always pair it with black pepper, which increases curcumin absorption significantly through piperine. For therapeutic purposes at the doses used in clinical trials, a bioavailability-enhanced supplement is more practical than food alone.

10. Walnuts

Walnuts are unusual among plant foods because they combine three liver-relevant compounds in a single food: ALA omega-3 fatty acids, ellagitannin polyphenols, and arginine, an amino acid that supports nitric oxide production independently of the nitrate pathway.

The most rigorous trial data for walnuts in fatty liver disease comes from the DIRECT PLUS trial, conducted by Dr. Anat Yaskolka Meir, Dr. Iris Shai, and colleagues at Ben-Gurion University of the Negev, published in Gut in 2021. This 18-month randomized controlled trial enrolled 294 adults with abdominal obesity and dyslipidemia and assigned them to three groups: a healthy dietary guidelines group, a Mediterranean diet group, and a green-Mediterranean diet group. Both Mediterranean diet groups included 28 grams of walnuts daily. Liver fat was measured by proton magnetic resonance spectroscopy.

After 18 months, the standard Mediterranean diet group with walnuts reduced intrahepatic fat by 19.6%, compared to 12.2% in the healthy dietary guidelines group. The green-Mediterranean group went further still, achieving a 38.9% reduction, but that group also consumed three to four cups of green tea daily and 100 grams per day of Mankai duckweed as a green shake, so the additional benefit beyond the standard Mediterranean diet belongs to that combination of polyphenol-rich additions, not to walnuts alone. What the trial clearly establishes is that a walnut-containing Mediterranean diet significantly outperformed standard healthy eating guidance in reducing liver fat, and that greater walnut intake was independently associated with greater liver fat loss in the study analysis. The study also notes the additive effect of eating more greens at the same time. 

There are a few mechanisms at work. ALA from walnuts is converted to EPA, which supports anti-inflammatory pathways in liver tissue. Ellagitannins are converted by gut bacteria to urolithin A, which has demonstrated anti-inflammatory effects. And the arginine content supports nitric oxide production in liver sinusoidal endothelial cells, which, as noted in the leafy greens section, is specifically impaired in fatty liver disease.

How to use it: 28 grams, roughly a small handful or about 14 walnut halves, is the dose used in DIRECT PLUS. Raw walnuts are preferable to roasted, as heat can oxidize the ALA. They work well added to salads, oatmeal, or eaten as a snack alongside other polyphenol-rich foods.

What to Stop Eating: The Foods That Cause Fatty Liver

We’ve covered very thoroughly what foods to include, but you need to look at the food list below and eliminate them if you want to make really good progress. Put in the good and kick out the bad. 

Remember, the way to beat fatty liver disease is to eat real foods. 

• Added sugars and fructose. High-fructose corn syrup and added sugars drive de novo lipogenesis in the liver, meaning the liver converts excess fructose directly into fat. Sugary drinks are particularly damaging. This is priority number one. 
• Ultra-processed foods. These are products with more than five ingredients that include refined oils, added sugars, emulsifiers, and artificial additives. The ultra-processed food category alone is the single strongest dietary predictor of NAFLD in large epidemiological studies. This is priority number two. 
• Refined carbohydrates. White bread, white rice, pasta, crackers, and pastries spike blood sugar and insulin, which signals the liver to produce and store fat.
• Red and processed meat. High saturated fat intake is directly associated with worsening liver steatosis. Processed meats also deliver nitrites and advanced glycation end products that increase hepatic inflammation.
• Alcohol. Even moderate alcohol consumption accelerates liver inflammation and fibrosis in people who already have fatty liver disease. At Hallelujah Diet, we recommend none.

A Hallelujah Diet Perspective

There’s no approved drug to help you with fatty liver disease. The European American Liver Guidelines agree that lifestyle modification, especially your diet, is the primary way to conquer it.

This isn’t just something you do while waiting for something else to come along that will actually work. This is the intervention that reverses fatty liver disease. 

The Hallelujah Diet simultaneously addresses the negative drivers of fatty liver disease and provides the positive foods that promote liver health. Our diet is naturally high in fiber, and polyphenols. We don’t have added sugars, refined carbohydrates, alcohol, or processed foods. Everything from leafy greens, extra-virgin olive oil, to legumes, oats, barley, flax seeds, walnuts, omega-3 fish oil, garlic, turmeric, and maybe even black coffee are all part of the Hallelujah Diet. Well, maybe not the coffee, but we’re not so against it anymore. The Hallelujah Diet is indeed very healthy for your liver. 

Along with the whole foods plant-based diet, we add a couple of things to make it even better:

1. BarleyMax for the concentrated micronutrients and the liver-supportive antioxidants.
2. Fish oil for long-chain EPA and DHA at therapeutic levels.
3. Professional Strength Curcumin for its anti-inflammatory properties that have been proven over and over in clinical trials.
4. B-Flax-D for the flax fiber, lignans, and omega-3 ALA, along with a little bit of vitamin D, vitamin K2, B12, B6, and zinc.

 

These additions fill gaps that even an excellent whole foods diet leaves open. 

If you have been diagnosed with fatty liver disease, or if you suspect you have it based on metabolic risk factors like elevated triglycerides, high fasting insulin, or abdominal obesity, this is your roadmap. The liver wants to heal. God designed it to heal, too. Your job is to get out of its way, give it the right building blocks, and enjoy the good health that God desires you to have. 

References

1. Ryan MC, Itsiopoulos C, Thodis T, et al. The Mediterranean diet improves hepatic steatosis and insulin sensitivity in individuals with non-alcoholic fatty liver disease. Journal of Hepatology. 2013;59(1):138-143. https://pubmed.ncbi.nlm.nih.gov/23485520/

2. Liu, C., Liu, Y., Liu, J. et al. Associations between intake of different types of vegetables and metabolic dysfunction-associated fatty liver disease: a population-based study. BMC Public Health 25, 315 (2025). https://doi.org/10.1186/s12889-025-21331-4 

3. Li H, Wang X, Ye M, et al. Does a high intake of green leafy vegetables protect from NAFLD? Evidence from a large population study. Nutr Metab Cardiovasc Dis. 2021;31(6):1691-1701. https://doi.org/10.1016/j.numecd.2021.01.009  

4. Nigam P, Bhatt S, Misra A, et al. Effect of a 6-month intervention with cooking oils containing a high concentration of monounsaturated fatty acids (olive and canola oils) compared with control oil in male Asian Indians with nonalcoholic fatty liver disease. Diabetes Technology and Therapeutics. 2014;16(4):255-261. https://pubmed.ncbi.nlm.nih.gov/24625239/

5. Rezaei S, Akhlaghi M, Sasani MR, Barati Boldaji R. Olive oil lessened fatty liver severity independent of cardiometabolic correction in patients with non-alcoholic fatty liver disease: a randomized clinical trial. Nutrition. 2019 Jan;57:154-161. https://pubmed.ncbi.nlm.nih.gov/30170304/
   
   
6. Pintó X, Fanlo-Maresma M, Corbella E, et al.; PREDIMED Study Investigators. A Mediterranean diet rich in extra-virgin olive oil is associated with a reduced prevalence of nonalcoholic fatty liver disease in older individuals at high cardiovascular risk. Journal of Nutrition. 2019;149(11):1920-1929. https://pubmed.ncbi.nlm.nih.gov/31334554/
   
   
7. Wu, X., Tjahyo, A.S., Volchanskaya, V.S.B. et al. A legume-enriched diet improves metabolic health in prediabetes mediated through gut microbiome: a randomized controlled trial. Nat Commun 16, 942 (2025). https://doi.org/10.1038/s41467-025-56084-6 

8. Ho HBT, Piper S, Sherrill C, et al. Effects of oat beta-glucan on LDL-cholesterol and non-HDL-cholesterol: a systematic review and meta-analysis of randomized controlled trials. European Journal of Clinical Nutrition. 2016;70(11):1239-1245. https://pubmed.ncbi.nlm.nih.gov/27273067/
   
   
9. Yari Z, Rahimlou M, Eslamparast T, et al. Flaxseed supplementation in non-alcoholic fatty liver disease: a pilot randomized, open labeled, controlled study. International Journal of Food Sciences and Nutrition. 2016;67(4):461-469. https://pubmed.ncbi.nlm.nih.gov/26983396/
10. Wu SF, Wang XC, Qi W, et al. Flaxseed powder supplementation in non-alcoholic fatty liver disease: a randomized controlled clinical trial. Food and Function. 2025. https://pubmed.ncbi.nlm.nih.gov/39878023/
    
    
11. Rezaei S, Akhlaghi M, Sasani MR, Barati Boldaji R. Flaxseed oil in the context of a weight loss programme ameliorates fatty liver grade in patients with non-alcoholic fatty liver disease: a randomised double-blind controlled trial. British Journal of Nutrition. 2020. https://pubmed.ncbi.nlm.nih.gov/31992372/
    
    
12. Scorletti E, Bhatia L, McCormick KG, et al.; WELCOME Study. Effects of purified eicosapentaenoic and docosahexaenoic acids in nonalcoholic fatty liver disease: results from the WELCOME study. Hepatology. 2014;60(4):1211-1221. doi: 10.1002/hep.27289. PMID: 25043514. https://pubmed.ncbi.nlm.nih.gov/25043514/
    
    
13. Song L, Zhao XG, Ouyang PL, et al. Combined effect of n-3 fatty acids and phytosterol esters on alleviating hepatic steatosis in non-alcoholic fatty liver disease subjects: a double-blind placebo-controlled clinical trial. British Journal of Nutrition. 2020;123(10):1148-1158. doi: 10.1017/S0007114520000495. PMID: 32054543. https://pubmed.ncbi.nlm.nih.gov/32054543/
    
    
14. Ruhl CE, Everhart JE. Coffee and caffeine consumption reduce the risk of elevated serum alanine aminotransferase activity in the United States. Gastroenterology. 2005;128(1):24-32. doi: 10.1053/j.gastro.2004.09.075. PMID: 15633120. https://pubmed.ncbi.nlm.nih.gov/15633120/
    
    
15. Grazioli E, Tranchita E, Borriello G, et al. Effect of coffee consumption on non-alcoholic fatty liver disease incidence, prevalence and risk of significant liver fibrosis: systematic review with meta-analysis of observational studies. Nutrients. 2021;13(9):3230. doi: 10.3390/nu13093230. PMID: 34578919. https://pubmed.ncbi.nlm.nih.gov/34578919/
    
    
16. Sangouni AA, Mohammad Hosseini Azar MR, Alizadeh M. Effect of garlic powder supplementation on hepatic steatosis, liver enzymes and lipid profile in patients with non-alcoholic fatty liver disease: a double-blind randomised controlled clinical trial. British Journal of Nutrition. 2020;124(5):450-456. https://pubmed.ncbi.nlm.nih.gov/32312333/

17. Panahi Y, Kianpour P, Mohtashami R, et al. Efficacy and safety of phytosomal curcumin in non-alcoholic fatty liver disease: a randomized controlled trial. Drug Research. 2017;67(4):244-251. https://pubmed.ncbi.nlm.nih.gov/28158893/
    
    
18. He Y, Chen X, Li Y, et al. Curcumin supplementation alleviates hepatic fat content associated with modulation of gut microbiota-dependent bile acid metabolism in patients with nonalcoholic simple fatty liver disease: a randomized controlled trial. American Journal of Clinical Nutrition. 2024;120(1):66-79. https://pubmed.ncbi.nlm.nih.gov/38795741/
    
    
19. Yaskolka Meir A, Rinott E, Tsaban G, et al. Effect of green-Mediterranean diet on intrahepatic fat: the DIRECT PLUS randomised controlled trial. Gut. 2021;70(11):2085-2095. https://pubmed.ncbi.nlm.nih.gov/33461965/